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The observed range of signaling outputs to TGF- β ligand in different cell types and under different conditions can be explained with differences in cellular protein concentrations and with changes in effective rate constants due to cross-talk with other signaling pathways.
These differences may be explained with differences among individual hPSC lines, supporting the necessity of isogenic disease models.
Increased percentages of HER2/neu negative cells than expected can be explained with differences of cell proliferation rates in in vitro and in vivo.
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Fifty-five PC1cent (PC1) of the morphological differences may be explained through a difference in transverse dimension in the lateral side of the palate (narrower for oral breather and larger for normal breathers Fig. 3) and in the height of the palatal vault, while 15% (PC2) of the differences may be explained with a difference only in the vertical dimension.
The difference in this study, between female and male was only one fourth of the difference between the work group and sick leave group, which indicated that work support in the work group not could be explained with gender differences, as we tend to do.
It can be explained with a difference in the methodology of genome sequencing and assembly [ 80, 81].
So, the differences could be explained with slightly different values of the PEP/pyruvate ratio for growth on the same substrate.
The different magnitude of the photocurrents obtained with [Fe CN 6]4− and Fe(II) can be explained with the known difference in the respective rate constants of electron transfer without having to postulate adsorption.
We show that this aspect of the fovea-parafovea difference
Discrepancies can be explained with tree reconstruction artifacts and differences in genome evolution.
Some of these differences can be explained with the fact that the schemes have been developed for different use cases (e.g. for data warehouses [ 66] as compared to research networks [ 20]).
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