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These transposon-free regions (TFRs) cannot be explained using random models of transposon insertion, and are often associated with genes that play important roles in development, such as members of the HOX, SOX, FOX and TBX gene families [ 8, 9].
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At level 2 of the model, the random parameters from model level 1 were explained using a variance analysis model with fixed effects.
Each line is explained using line comments.
If heterogeneity could not be explained, we used the random effects model to assess the robustness of the results to the choice of model.
When a substantial amount of statistical heterogeneity was found (when I2 > 50% [ 12]) and could not be explained, a random effects model was used.
Minor differences could be explained by random uncontrolled fluctuations.
The ethnically related distribution of ADH1B cannot be explained only by random genetic drift.
The random jumps above the target BER line can be explained by the use of BPSK even when the ppSNR fell in the outage region (see Fig. 2).
Temporal changes in allele frequencies could not be explained by genetic drift or random sampling.
If heterogeneity was detected and could not be explained by subgroup or sensitivity analyses, we used the random-effects model or did not pool results.
When there was statistically significant heterogeneity within groups that could not be explained by subgroup or sensitivity analyses, we used a random-effects meta-analysis model.
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