This was explained firstly by the fact that ISS and GIAD scales were dedicated to the Internet, so they included other elements beside gaming, whereas DAS was specific to online video games.
Material selection guide line is explained firstly both for main parts and wearing parts.
This may be explained by, firstly, both PET-CT studies included in this meta-analysis were of small sample size and, secondly, because sensitivity results in chemonaive patients were also rather low in both studies.
Not ideal fitting of the experimental curves by the theoretical ones can be explained by some reasons: firstly, we assume that all our particles have oblate shape that is not evident exactly; secondly, we used the averaged radius; and last, as it was shown in our previous work [9], peak of surface plasmon is very sensitive to material of the host matrix.
This can firstly be explained by the fact that some of the biggest airlines of the world operate under a license granted by one of these Member States mentioned above, e. g., British Airways, Lufthansa, Air France/KLM, etc.
This difference could be explained by two observations: firstly, patients assuming analgesic mixtures are historically older, when the clinical practice for treatment of chronic headache was based on the use of classic mixtures, as association of caffeine, phenothiazines or barbiturates and NSAIDs.
These differences can be explained by two factors: firstly, a significant difference in the frequency of polymorphic genotypes in different populations and, secondly, difference in AF etiology.
The mechanism of the PL enhancement could be explained by following two reasons: Firstly, we propose that the electron-donating effect which resulted from the abundant amino acid groups on the surface of RNase A, especially those amino acids with benzene rings, might contribute a lot to the much enhanced PL intensity of the C-dots.
This may be explained by the following two mechanisms: firstly, it is due to the increased production of subretinal fluid secondary to a persistent subclinical active inflammation, and secondly, it is due to the decreased absorption of the subretinal fluid by the malfunctioning of the RPE pump and loss of the RPE cells due to chronic inflammation.
Firstly, they could be explained by an increased rate of de novo mutations among individuals with AUDs, confirming our hypothesis.
