First, low co-morbidity figures may be explained by under-diagnosis of psychiatric disorders because not all children were thoroughly screened by child psychiatrists.
Although this might be explained by under-reporting of non-conventional pathways, previous studies using the WHO Encounter Form have successfully identified non-conventional pathways [ 6, 28].
Third, undertreatment of the increased CV risk in RA by PCPs might be explained by under-recognition because CRFs were assessed more frequently in diabetes in comparison with RA.
Although insufficient food intake (true under-nutrition) due to poor appetite and dietary restrictions contributes to these problems, there are features of the syndrome that cannot be explained by under-nutrition alone.
This may be explained by under-recording of certain comorbidities in people with dementia, as they may be less likely to be investigated than other people with chronic diseases.
First, it is hard to believe that the largely absent CV-RM by rheumatologists is explained by under-recognition because the increased CV risk in RA must presently be well known among rheumatologists.
Morbidity indicators i.e., incidence or prevalence measures, (M in Table 2), require accurate disease detection, with KC greater than 0.4, or K1 > 0.4, if false positives are explained by under-coded diagnoses.
This can be explained by near optimal nutrient provision under conditions in which light and CO2 are not limiting low cell concentrations and thin cultures [ 28].
In fact, we found that in lung cancer over expression of BCL2 could be explained by the under expression of MIR15A, MIR16-1, and MIR16-2, whine in kidney tumours over expression of DFFB, HTATIP and RELA could be related to the under expression of MIR124A-1, MIR124and, and MIR124A-3 (Table 5).
