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Focusing on face centered cubic (fcc) metals, simulations suggested that the increased strength can be explained by truncated spiral dislocation sources, 6– 8 as was confirmed by a number of in situ transmission electron microscopy (TEM) experiments.
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This clinical difference might be explained by the length of truncated proteins identified in each patient.
The presence of a nonsense mutation for W460 in an LINCL patient can be explained by premature termination and a truncated protein devoid of activity, rather than a direct involvement of this W mutant in any critical function in the TPPI structure.
Thus, the 153 154 bp DNA fragments that are visible in the gel may be explained by the presence of multiple, predominantly truncated, copies of this transposon representing the remnants of the Helitron amplification process that occurred at some point in the virilis group evolution.
The differences were too big to be explained by randomness.
The point may perhaps best be explained by example.
Complicated procedures can be explained by an embedded video.
This might be explained by several factors.
This can be explained by two reasons.
This could be explained by travel motives.
This may be explained by several reasons.
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