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This can be explained by suggesting that the residual SFM value for true audio signals is primarily determined by the (low-power) harmonics which are modeled as noise components instead of tonal components.
The discrepancy of correlation lengths in the mapping of ZnO nanowire could be explained by suggesting that the ZnO is consisted of a collection of crystalline grains due to structural defects and the grain size decreases (i.e., the defect density increases) along the growth direction.
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This temporal relationship was also documented in other studies[3, 48, 50], and has been explained by suggesting a close mechanical dependence between arterial pressure and the derivative of respiratory volume, i.e., the respiratory flow[3].
This surprising result was explained by suggesting that the Mn3+ active species might be regenerated in the reactant stream through the surface oxidation of Mn2+ ions by NO, or N2O.
The lack of inhibition in this case may perhaps be explained by findings suggesting that estrogen is an inhibitor of np2 expression [31].
However, 88% of the variation for T1.10 can be explained by T1.04, suggesting that developmental variation is mainly due to heterogeneity at the initial stage.
This discrepancy may partially be explained by evidence suggesting that glutamate receptor activation/blockade can have different effects at different developmental stages (Tashiro et al., 2003, Wong and Wong, 2001).
In Table 4 some negative results are reported: the sub-optimal codes can all be explained by chance, suggesting that it is not only the composition of amino acids that matters, but also the assignment to proto-adapters.
Our finding strongly indicated that more than 81% (r 2) of the variation in Alu density could be explained by CI frequency, suggesting that Alus may play an important functional role in intra-domain interactions.
Surprisingly, we find that redundancy within complexes cannot usually be explained by gene duplication, suggesting alternate buffering mechanisms.
It has been estimated that 90% of cancer deaths and half of heart disease mortality cannot be explained by genetic factors, suggesting an environmental origin.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com