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The reduction in tubular apoptosis in TLR2−/− mice can also be explained by studies demonstrating a proapoptotic function of TLR2 [48], [48].
This may be explained by studies that have reported marriage as a psychosocial correlate for mental illness in Pakistan as discussed above.
This discrepancy in the risk of developing diabetes may be explained by studies on the pathogenesis of type 2 diabetes in people with familial risks.
Additionally, the high levels of HIV-RNA in herpetic lesions from dually infected patients [ 38] may be explained by studies in vitro demonstrating that HSV-2 increases HIV transcription, which supports the higher infectivity in co-infected individuals.
These seemingly contradictory results may be explained by studies that showed that P-gp is an efficient methotrexate transporter in cells that were deficient in the methotrexate carrier [ 46, 47].
This may in part be explained by studies which have shown that isolation failure from suspected cholera stools by stool culture during acute diarrhoea outbreaks may be explained by the inactivation of V. cholerae by in vivo vibriolytic action of the phage and/or nonculturability induced as a host response [ 26].
Similar(52)
The differences might be explained by study designs and environmental factors involved.
This can be explained by studying how the electron rays are deflected by the morphology at t = 15 min as shown in Fig. 6a.
This observed variation can be explained by studying the accompanying PC loading line plots.
Heterogeneity could not be explained by study design, differences in population/interventions/outcomes.
This result can be explained by studying the elephant shark genome.
More suggestions(18)
be elucidated by studies
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be illustrated by studies
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be supported by studies
be explained by economics
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be assessed by studies
be followed by studies
be provided by studies
be tested by studies
be detected by studies
be set by studies
be explained by referees
be demonstrated by studies
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Justyna Jupowicz-Kozak
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