The disparity cannot be explained by any apparent biological factors.
This sequential response can be explained by an apparent sudden release of nutrients consequent to arthropod death that triggered a series of responses in the microbial loop.
The relative energy intake was not different, which can be explained by the apparent significant increase of body mass during the competition phase, and is likely an artifact of the aggregation of data from various studies.
The good performance in general can be explained by the apparent difference between actives and inactive compounds as seen during the clustering, and the better performance on the test set compared to the validation set could be due to slightly higher nearest neighbour similarity to the training actives (0.53 for test actives and 0.48 for validation actives).
We suggest, however that this observation may be explained by the apparent lack of sigS expression in SH1000.
The lack of a significant 2nd chromosome QTL in the F2 BLUP dataset can be explained by the apparent equal and opposite non-additive effects of the two QTL found in the sons dataset.
Examining the 18 genes with the lowest R2 values (less than 0.8) revealed a non-linear relationship that can be explained by an apparent saturation at the 50× concentration of RNA (Fig. 1C).
This may be explained by an apparent upper limit in song rate, since the highest mean group song rates observed in this study are in the upper range of what has been observed in other studies using male starlings, both in the wild and in captivity [13], [25], [26], [27].
The relative promiscuity of the second PDZ domain can be explained by its apparent reliance on hydrophobic interactions within the positional window "−4 to −7" and, likely, at the position "−1", which makes this domain less sensitive to lysine/arginine versus aspartate/glutamate compositional biases, thus allowing for a much greater variety of amino acids acceptable at these positions.
Inhibition of NF-κB-mediated gene transcription by mutant, polyglutamine repeat expanded ataxin-7 can therefore be explained by its apparent ability to inhibit ubiquitin-dependent proteolysis.
