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Reduced function of skeletal muscles [ 13] might be due to functional changes in cortical motor areas [ 14], but could also be caused by changes in muscle metabolism due to increased oxidative stress [ 15, 16].
The correlations could be due to functional polymorphisms or to population structure.
The deterioration of the regenerative potential upon aging might be due to functional changes in adult stem cells.
Since sequences of dmNnf1R-1 and dmNnf1R-2 are very similar, we thought that the lack of a dmMis12-like phenotype may be due to functional redundancy.
This relatively weak phenotype, i.e. lessened seedling growth, in response to tunicamycin may be due to functional compensation by AtCRT1b that is present in the mutant.
Considering that this could be due to functional redundancy with SseK3, which was functional in the strains tested by Kujat Choy et al. [12], we initially focussed our efforts in studying a strain mutated for all three SseK family members.
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The role of Nef in anti-apoptotic function is due to functional interaction with ASK-1 which links both the Fas- and the TNFR- mediated signals (by Fas ligand and TNFα) to the downstream JNK/p38 pathway [6], [24].
Within the aerobic scope the adjustments are due to functional variation.
Many breakwaters are, due to functional requirements, designed for small wave overtopping discharges.
The decrease in FMZ BPND may be related to the loss of neuronal integrity, and it seemed that the decrease in CMRO2 in the regions showing no loss of neuronal integrity was due to functional hypometabolism [4].
Western Blot analysis revealed expression of all constructs (Fig. 3), suggesting that the differences in activity are due to functional differences and not due to lack of expression.
More suggestions(15)
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