Sentence examples for be differences in disease from inspiring English sources

Furthermore, pathogen virulence can respond positively or negatively to a range of variables, such as temperature, nutrient availability, or habitat quality [4] [6]; changes in environmental conditions can promote physiological stress that impairs host immunity [7] [9], and there may be differences in disease susceptibility between host genotypes [10], [11].

The reasons for the discrepancy could be differences in disease induction, route of dosing and dose, used substrains of mice and the animal housing environment.

Explanations for contradictory findings between these two studies and our study may be differences in disease activity and disease duration in the examined study cohorts.

The sample size was independently powered for the three age strata, with 400 eligible children from the 0 11 months age group and 370 eligible children from each of the two older age strata (12 23 and 24 59 months), because there could be differences in disease incidence and care-seeking patterns between younger and older children.

Although both models are formatted to address barriers to effective and efficient health care, there are differences in disease prevalence and functional performance between groups.

Here we used known vsp DNA sequences encoding for B. turicatae and Borrelia hermsii Vsp proteins with variable regions and then studied whether there are differences in disease expression and tissue localization of their corresponding serotypes during mouse infection.

Although all five were able to infect Brussels sprout leaves (Table 1), there were differences in disease progression between isolates.

26 Unexpected issues in the current study were differences in disease duration and baseline severity between the two treatment groups, which were due to chance.

Physicians have long known that there were differences in disease susceptibility and drug response among ethnic groups, [ 32- 38] between the sexes, [ 39- 43] and even between monozygotic twins [ 67- 70].

In addition, the clinic patient group was more symptomatic than the pre-IPT screening group and there are likely to have been differences in disease spectrum between these groups.

The exact mechanisms accounting for differences in the magnitude of low- versus high-STZ dose effects are not fully known, but our data indicate that there are differences in disease onset (based on earlier blood glucose elevation in high dose mice) and metabolic/catabolic effects (a greater loss of body and fat pad mass in high-dose mice) between the two dosing regimes.