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Because endoleaks have variable flow rates, they can be detected at variable times after contrast material injection.
The MDR1 mRNA was not detected after 22 cycles of PCR in the 10 gastric cancer cell lines but could be detected at variable levels after 35 cycles of PCR with the exception of SNU-16, suggesting a significantly low level of MDR1 mRNA expression in the gastric cancer cells tested.
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C/EBPβ mRNA expression was found to be elevated in 100% of mammary tumors isolated from these ErbB2 overexpressing mice and, interestingly, C/EBPα mRNA was detected at low levels in 30% of tumors, C/EBPδ mRNA was detected at variable levels in 50% of the tumors, and CHOP mRNA was detectable in most of the tumors examined [ 26].
In contrast, chlorine and oxygen were detected at variable levels in almost all layers.
Moreover, KSHV DNA was detected at variable levels in DNA extracted from culture supernatants, suggesting that a quote of KSHV lytic infection may spontaneously occur in these cells.
After application of a Bonferroni correction (by sample), no significant strand bias was detected at variable sites in Illumina samples.
As shown in Figure 1, the immunoreactivity of AT1R was detected at variable levels, and was localized both on the membrane and in the cytoplasm of tumour cells.
As shown in Figure 2A F, the immunoreactivity of IDO was detected at variable levels, and was localised in the cytoplasm of tumour cells.
As shown in Figure 2A C, GnT-V immunoreactivity was detected at variable levels, and was found in the cytoplasm of cancer cells, which were identified using PCNA co-staining.
On western blotting using paired LYO and MYO from 11 untreated patients, TIMP2, VEGFA, and FBLN5 were detected at variable levels with increased TIMP2 and FBLN5 and lower VEGFA productions, at least in seven out of 11 LYO as compared with paired MYO (Fig. 6D).
The reduction in heritability estimated for the combined CA and NM cohort, in comparison to the individual population estimates, is a complex phenomenon that likely reflects environmental differences as well as differences in the host genetic basis for differential susceptibility to a variety of BRDC pathogens that were detected at variable frequencies within the two populations.
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