While replication of Lp in cultured murine BMDM is controlled through activation of the NLRC4/NAIP inflammasome, the role of the inflammasome in mediating resistance to Lp in alveolar macrophages and in the lungs of whole animals is only partially understood.
The activity of a MAPK in cells is controlled through phosphorylation activation by its upstream kinases, MAPKK and MAPKKK, and dephosphorylation inactivation by its negative regulator, MAPK phosphatase/s.
The survival of animal tissues and organs is controlled through both activation and suppression of programmed cell death.
The core NO· specific response (Fzf1p early and late response clusters), unlike other transcriptional responses, was controlled through the activation of the transcription factor Fzf1p. NO· also triggered a general environmental stress response, which was shared by exposing to oxidative agents.
We also examined whether COX-2 expression was controlled through ligand-mediated activation of PPAR γ in ovarian carcinoma cells.
To examine whether COX-2 expression is controlled through PPAR γ activation in ovarian carcinoma cells, OVCAR-3 cells and the ovarian carcinoma cells established at our hospital were cultured in a semiconfluent state in normal culture medium or in media supplemented with 0.1 20 μ M 15d-PGJ2, a PPAR γ activator as described in Materials and Methods.
After completion of activation, flow rate of carrier gas was controlled through a mass flow controller at 40 mL/min, an optimum rate from previous studies [19].
The activation of MPF is controlled through the dephosphorylation of CDC2 at tyrosine15 [ 25].
Curiously, while TGF-β1 is considered an anti-inflammatory cytokine, it induces leukocyte recruitment.[8], [9] TGF-β1 activity is controlled predominantly through activation of the latent molecule.
Chemotaxis is a crucial event for neutrophil migration to local infection and is controlled mainly through activation of G-protein-coupled receptors.
