Exact(3)
The unspecific signals could be caused by binding of non-target proteins by the antibody.
Thus, lactational transfer of PFAAs, including perfluorooctanoic acid (PFOA), perfluorooctane sulfonate (PFOS), perfluorononanoic acid (PFNA), and perfluorohexane sulfonate (PFHxS), is believed to be caused by binding to milk proteins (Butenhoff et al. 2006; Fromme et al. 2010; Kärrman et al. 2010).
The nuclear import defect at low concentrations of PF74 may be caused by binding of this compound to a reactive groove on the viral capsid, leading to competitive inhibition of capsid interaction with the host proteins Nup153 and CPSF6 (Matreyek et al., 2013; Price et al., 2014).
Similar(57)
Using mutant forms of Ras and Raf-1 we could show that the reduced kinase activity of RBD MEKK1 fusion proteins was caused by binding to the Ras protein.
SPR sensorgrams include real-time information on the changes in mass that are caused by binding in a bimolecular interaction, such as that between probe [P] and target [T], as follows [38, 39].
We suspect that this degradation is caused by binding of mir-202-3p mir-202-3p mir-202-3pes in toe theget mRNAs, leadidentifiedir degradation by the RIsites
First, it is caused by binding of wall teichoic acids to the C-6 position of NAM (Bera et al. 2007).
Whether the positive staining of neutrophils and MPO+ precursor cells is due to autocrine secretion or is caused by binding of IL-17 at the IL-17 receptor could not be determined by our staining.
Both effects are caused by binding of AMP at two exchangeable sites on the γ subunits and are opposed by ATP, which binds at the same sites (Scott et al., 2004; Xiao et al., 2007).
Transactivation is caused by binding of two activated glucocorticoid-cytosolic glucocorticoid receptor complexes as a dimer to the promotor of glucocorticoid-regulated genes, which leads to up-regulation of the synthesis of certain regulatory proteins by transcriptional activation.
To exclude the possibility that down-regulation was caused by binding of TFO−C141S to the TFS 4MM site present in p4-1, plasmids p15-2 and p11-1 were treated with TFO−C141S and transfected into SKOV3 cells.
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