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Overall, these experiments showed that ISP stimulation at sufficient stimulus intensity (4.5 6 mA) can bring about stimulus phase-dependent amplitude modulation of the EEG, an effect which cannot be attributed to peripheral sensory or arousal effects.
Despite the mechanisms that generate the metabolic disorders are complex and multi factorial, they could be attributed to peripheral shunting, leading to decreased oxygen extraction in a heterogeneous microcirculatory compartment [7,42,43].
Most symptoms can be attributed to peripheral effects.
We speculated that this phenomenon may be attributed to peripheral vasoconstriction.
The authors therefore suggested that the single shift in behavioral response could be attributed to peripheral, not central, pathways [ 21].
Lower T4 levels associated with HCB exposure may be attributed to peripheral disposition of T4 (Kleiman de Pisarev et al. 1989) or increased hepatic T4 metabolism (Kleiman de Pisarev et al. 1990).
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Pain in knee osteoarthritis (OA) has historically been attributed to peripheral pathophysiology; however, the poor correspondence between objective measures of disease severity and clinical symptoms suggests that non-local factors, such as altered central processing of painful stimuli, also contribute to clinical pain in knee OA.
Primary hyperalgesia occurs within the area of tissue injury and has been attributed to peripheral release of inflammatory mediators that increase the sensitivity of primary nociceptive fibers to noxious stimuli (for review see [59]).
In DPN patients, such alterations have been attributed to peripheral nerve deterioration and related sensorimotor complications (3, 21– 21).
Kroenke et al [ 7] found in their systematic review that dizziness was attributed to peripheral vestibulopathy in 44%, central vestibulopathy in 11%, psychiatric causes in 16%, other conditions in 26%, and an unknown cause in 13% of cases.
The modulation of the size of the limb can alter subjective feelings of pain and motor imagery can cause an increase in pain and swelling that cannot be attributed to increased peripheral sensory afference arising from nociceptors or peripheral neural injury [ 142, 150].
More suggestions(16)
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