In accordance with previous research done by Pajchel et al. [34], 31P BD NMR signals were fitted using two lines: a narrow one, which corresponds to the nuclei of phosphorus-31 originating from inside the crystal, and a broad one, which may be attributed to the nuclei of phosphorus-31 located in the hydrated surface layer.
However, Kulesza (2008) located this cell group at a position which could be attributed to the medial periolivary nucleus or medioventral periolivary nucleus, a position in which cells have been also noted by Bazwinsky et al. (2003) by the use of immunohistochemistry.
This phenomenon could be attributed to the high rate nuclei and therefore with the formation of more silver nanocrystals [52].
The midbrain accordingly does not contact the pons at all, and, as we have seen, only a minor rostral part of the dorsal raphe nucleus can be attributed to the caudal midbrain (Puelles et al. 2012b; Allen Developing Mouse Brain Atlas; http://www.developingmouse.brain-maps.org).org
The nature of PTEN localization in the nuclei can be attributed to binding by one ubiquitin modification, which also produces a delay in electrophoretic mobility.
The apparently stable Bcd intensity within a given nucleus could either be attributed to the lack of significant change in Bcd intensity over the majority of the anterior half of the embryo, or to Bcd being trapped within the energid (surrounding cytoplasm of a nucleus), even during a nuclear division cycle.
Many tractography maps also included connections that have not been previously observed (e.g., callosal connections from the lateral geniculate nucleus), which could be attributed to false positives, or to the fundamental differences between connectivity mapping techniques.
An exception from this behavior can be found for cell 2C showing a homogeneous cell nucleus, which might be attributed to the fact that DCT is only effecting cells during mitosis.
As shown in Figure 3 k), it decreased at higher grades, which is indicative of lowering intensity in HG nuclei that may be attributed to the damage of P63 receptors at high grades.
This may again be attributed to the dismantling of the high grade nuclei and to the subsequent destruction of P63 receptors.
