Sentence examples for be attributed to activation from inspiring English sources

It potentially could be attributed to activation in S1 and S2 and recorded together, since both are activated by noxious stimuli [13].

This might be attributed to activation of Wnt/Ctnnb1 signaling, as shown by accumulation of non-phospho (active) Ctnnb1 and concomitantly increased expression of its target gene c-Myc (Fig.  6 and Additional file 5), a cell-cycle effector that contributes to G1 progression.

The enhancement in emission intensity with gradual increase in the applied voltage (Fig. 4) can be attributed to activation of more radiative recombination centres in ZnO NTs.

Collectively, suppression of KEAP1 expression in human NSCLC cells resulted in sensitization to chemotherapeutic agents, which may be attributed to activation of PPARγ and subsequent alterations in cell differentiation and CSC abundance.

Involvement of a region in the dorsal midbrain (Table 1 and Fig. 2a) was also detected in a fMRI study in rats and could possibly be attributed to activation of nociceptive bladder afferent nerves since measurements of immediate early gene expression in the brain after bladder irritation with cyclophosphamide identified this region [21].

Studies indicate that some pharmacological effects of resveratrol can be attributed to activation of the class III HDAC Sirt1 [22].

This insulin-dependent phosphorylation has been attributed to activation of the mitogen-activated protein kinase (MAPK) pathway, involving serine residues contained within the N-terminal ligand-independent activation domain (AF-1) of PPARα [23] and PPARγ [22].

The main components of marijuana including the tetrahydrocannabinol Δ-THC (1, Fig. 1) and other phytocannabinoids have long been known to exhibit analgesic activity, which has since been attributed to activation of the CB1R as well as the CB2R.

This was attributed to activation of Nrf2-ARE pathway, reduced NF κB activity, and increased oxygen availability via HIF1 signaling mechanisms.

Taken together, these results indicate that autophagy induced by silencing of EEF2K is attributed to activation of AMPK-ULK1 pathway, independent of the suppression of MTOR activity and stimulation of ROS production.

The increased lipid accumulation in Ces1g deficient mice was attributed to activation of hepatic SREBP1c processing leading to increased lipogenesis.