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Consequently, the failed Ca2+ response can be attributed to abnormal EC coupling rather than failure of action potentials.
Since dynamic epigenetic regulations occur during ICM formation and differentiation of primary germ layers, early embryonic lethality caused by Mbd3 deletion may be attributed to abnormal epigenetic modifications, and therefore dysregulation of gene expression in early embryos [30], [31].
Our main aim in this study was to investigate whether the abnormal Ca2+ responses can be attributed to abnormal action potentials or other factors such as failure of EC coupling.
To clarify whether these abnormal Ca2+ transients can be attributed to abnormal action potential generation or abnormal Ca2+ handling/excitation-contraction (EC) coupling, we developed a procedure to determine Ca2+ and action potential signals at the cellular level in isolated heart tissues.
Thus, D-AD pathogenesis may be attributed to abnormal activity of neurons and glial cells in the left ACG.
It is difficult to decide whether imaging results have to be attributed to abnormal processing of the stimuli or that the depressive state overrules brain processing.
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This was historically observed with the methods of clinical neurophysiology that revealed interictal deficient habituation of any kind of sensory responses - with the notable exception of the olfaction - that was attributed to abnormal thalamo-cortical interactions and its normalization during an attack.
The mechanism behind the formation and stability of these nanowires are attributed to abnormal faceting of the wetting layer [6, 13] or extended huts [10, 20], which is driven by the low surface energy of their {1 0 5} side facets under equilibrium phase.
Inattentiveness has been attributed to abnormal (deficient or excessive) phasic release of norepinephrine in the prefrontal cortex.
Using detection or discrimination measures or stimuli reported to trigger migraine (striped patterns or flickering lights) one finds differences between people with and without migraine, which is attributed to abnormal cortical processing in migraine, described by hyper- or hypoexcitability, heightened responsiveness, a lack of habituation and/or a lack of intra-cortical inhibition [50], [51].
Lineage infidelity correlated with failure of remission and was attributed to abnormal gene expression.
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