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An example for this case is a reaction transforming educt A into product B that can alternatively be activated by two activators E1 and E2.
The coagulation system can be activated by two pathways, the intrinsic and the extrinsic, of which both lead to the formation of the prothrombinase complex on phospholipid membranes.
In mammals, JNK is reported to be activated by two MAPK kinases (MAPKK) Mkk4 and Mkk7, with Mkk7 as the major MAPKK in TNF- or IL-1-induced JNK activation while both, Mkk4 and Mkk7, are required for stress induced activation of JNK [2].
EphA2 can be activated by two different mechanisms.
Proteasomes can be activated by two different types of regulatory complexes.
In previous sections we assumed that protein P can be activated by two mechanisms of different order.
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Complement system consisting of approximately 35 plasma and membrane-bound proteins can be activated by three different but partially overlapping routes: the classical pathway (CP), the alternative pathway (AP) and the lectin pathway (LP).
The complement cascade can be activated by four distinct pathways, three of which converge at the level of the C3 component, leading to the cleavage of C3 and C5 to their activated forms, C3a and C5a, as well as the formation of the terminal membrane attack complex [21], [22].
This pathway can be activated by three ligands: insulin, IGFI and IGFII.
The complement system can be activated by three different but overlapping classical, lectin and alternative routes [ 74].
The complement system may be activated by three different pathways: the classical, the lectin and the alternative pathway [ 3].
More suggestions(17)
be initiated by two
be stimulated by two
be implemented by two
be mediated by two
be ordered by two
be generated by two
be executed by two
be nominated by two
be governed by two
be afflicted by two
be performed by two
be addressed by two
be represented by two
be judged by two
be played by two
be accompanied by two
be succeeded by two
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