Sentence examples for bcl's from inspiring English sources

BCL's Panguna mine opened in 1972, three years before PNG was granted independence from Australia.

But according to a Jubilee Australia report last year, the vast majority on the island oppose BCL's return.

BCL's website quotes a legal magazine saying senior partner Ian Burton is widely thought of as "the finest criminal fraud lawyer by a long stretch … if there is a chance of nipping an investigation in the bud, he will do it".

However the significance of Bcl-xL's regulation of ceramide channel formation within cells was untested.

But the researchers discovered that Bcl-2's regenerative powers seem to reside in a different, unknown mechanism.

Due to Bcl-XL's anti-apoptotic function, we examined the role of ETK in regulating apoptosis in these cells.

Bcl-xL's mechanism of action is therefore a major component of chemoresistance in cancer cells [4].

As shown in Fig. 5B, neither deletion of the BH4 domain nor the Y101K mutation affected Bcl-xL's ability to interact with Praf2.

AKBA treatment blocked Bcl-xL's ability to increase levels of Slug, Snail and RelA RNAs (FIG. 5F), suggesting that Bcl-xL acts on these RNAs, as it does on the NF-κB responsive reporter, by increasing IκB kinase activity.

To examine Bcl-xL's effects on NF-κB activity in Xenopus, we used the NF-κB responsive p3XκB-Luc reporter plasmid together a mutated form of human IκBα (IκBsa) and acetyl-11-keto-β-boswellic acid (AKBA).

When co-injected with RNAs encoding Rel3 or Xp52, the active form of the NF-κB subunit protein Xp100 [87], RelAΔSP inhibited their ability to activate of the 3XκB-Luc reporter (FIG. 5G) and inhibited Bcl-xL's ability to increase RelA and Slug RNA levels (FIG. 5H), indicating that active NF-κB is required for Bcl-xL to induce increases in Slug and RelA RNA levels.