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Delayed activation of the naturally occurring process of programmed cell death during development of the peripheral nervous system has been suggested to represent the molecular basis of spontaneous regression in neuroblastoma.
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Finally, biological mechanisms at the base of spontaneous regression of the brain lesions are discussed.
Finally, we also debated the biological mechanisms at the base of spontaneous regression of the brain lesions.
To report the occurrence of spontaneous regression of three endometrial polyps detected by hysteroscopy.
[From the Introduction] This thesis is concerned with two aspects of spontaneous regression of retinoblastoma: 1) Retinoblastoma may lack TAF or has inhibitory factor to TAF and spontaneous regression occurs by vascular insufficiency; 2) Retinoblastoma cells undergo necrosis with an increased susceptibility when the calcium concentration is elevated.
Some cases of spontaneous regression have been reported, especially in young patients.
The 'true' incidence of spontaneous regression of neuroblastomas is uncertain.
Regression of colorectal cancer is exceedingly rare [ 9], and represents less than 2% of all cases of spontaneous regression [ 10].
However, the frequency of spontaneous regression is important when the benefits of screening procedures are considered.
The rate of spontaneous regression in CIN III lesions is controversial.
However, the molecular mechanism of spontaneous regression in NB remains unknown.
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