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Humans vary in their susceptibility to acquiring Staphylococcus aureus infection, and research suggests that there is a genetic basis for this variability.
RhoB has different effects on cell migration depending on the cell type and conditions, but the molecular basis for this variability is unclear.
The molecular basis for this variability is currently unknown but seems to be independent of the nature of the ABCD1 mutation; therefore the time of onset is unpredictable for pre-symptomatic patients.
The basis for this variability is unknown, but we observed a similar level of variation in another cell line that has a single copy of an unrelated GFP transgene: Jupiter (Karpova et al. 2006) (data not shown).
Identifying and understanding the neuroendocrine basis for this variability in rats and other species will be important for understanding the evolutionary physiology of reproductive strategies and for understanding genetic variation in brain function.
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The extent of functional recovery is highly variable among patients, and the basis of this variability is not known.
The basis of this variability in the RAP/PCWP ratio, and whether it conveys prognostic value, is not known.
Different patients can respond to the same NSAID in a variety of ways and the basis for this individual variability remains unclear.
In summary, all these results support a relatively minor contribution of the phenotypic plasticity to the ecotype differences observed in the proteome expression, suggesting an important genetic basis for the variability in gene expression in this species.
Although the substrate-dependent spin state regulation for some CYPs is well documented, the molecular basis for the variability in the substrate-free ferric spin equilibrium has not been determined.
We do not yet understand the basis for the variability, but assume that it represents a transient state and that these large cells begin to divide later than the others.
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