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The underlying basis for this model is the calculation of changes from known 'baseline ships'.
Using N-SYN to model PD pathogenesis offers two major advantages: N-SYN is found in ventral mesencephalic DA neurons of PD patients and is considered a critical factor in PD development [23], [25], thus providing a pathophysiological basis for this model; and, as we showed herein, nitrated α-synuclein affects DA neuron less severely and causes less acute lesions than 6-OHDA in vivo.
However, until now no legal and financial basis for this model does exist.
The findings of Maqbool, Saitoh et al. provide a new structural basis for this model.
Details of the calculation and basis for this model are included in File S1.
In this Review, we will discuss the basis for this model and its functional relevance.
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Although the results of this CSU SFES research study are informative, a national study of the SFES phenomenon would provide a stronger basis for understanding this model for transforming science education.
The results here provide a strong experimental basis for supporting this model and for discarding the idea that E. coli replication occurs in fixed replication factories.
On the basis of this model a formula for the concentration dependency of vacancies and adatoms on the potential was obtained.
Evidence for the neuro-anatomical basis of this model has come from patient studies.
On the basis of this model, t-tests for differential expression between each pair of sample types (leaf vs. bud, leaf vs. bud', and bud vs. bud') were conducted.
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