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Targeting transmembrane proteins - like E-cadherin - or increasing the levels of intracellular reactive oxygen species via enhanced activation of Rac1b are further mechanisms of MMP-induced EMT [38, 39]. Figure 1 Basic molecular changes underlying EMT.
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These processes are therefore potential regulators of the molecular changes underlying permanent pain states.
This article reviews the molecular changes underlying pancreatic neoplasms, with a special focus on the genetic changes that characterize the histologic types of pancreatic neoplasms.
After analyzing the molecular changes underlying hESC differentiation as well as their electrophysiological phenotypes, we then assessed the effect of hESC-CM transplantation on myocardial function.
The genetic and molecular changes underlying the disease are complex.
The molecular changes underlying MCN formation and progression are not entirely clear.
The molecular changes underlying epigenetics modifications include DNA methylation and histone modifications.
22– 24 Otherwise, however, little is known of the detailed biochemical and molecular changes underlying PTT dysfunction.
To understand the molecular changes underlying this process, we performed proteomic and RNA array analysis.
Here, we demonstrate key molecular changes underlying BPA-induced cellular reprogramming.
Diagnosis and management of this disease are complex, and the molecular changes underlying sensory cell impairment remain poorly understood.
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