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10– 15 Autophagy is the basic catabolic mechanism of cells to degrade unnecessary or dysfunctional cellular components through the actions of lysosomes, 16 which can ensure cellular survival during starvation by maintaining cellular energy levels.
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Autophagy or cellular self-digestion, the basic catabolic mechanism that involves degradation of unnecessary or dysfunctional cellular components through the actions of lysosomes, has been reported to be implicated in a broad spectrum of mammalian diseases.
Autophagy constitutes the basic catabolic mechanism that involves degradation of unnecessary or dysfunctional cellular components by lysosomes [ 12].
Autophagy is the basic catabolic mechanism that involves cell degradation of unnecessary or dysfunctional cellular components through the actions of lysosomes.
Autophagy constitutes the basic catabolic mechanism that involves the degradation of unnecessary or dysfunctional cellular components by lysosomes [ 12].
Autophagy is the basic catabolic mechanism that involves the cellular degradation of unnecessary or dysfunctional cellular components through the lysosomal machinery.
Genes responsible for the catabolic mechanism of ferulic acid were identified in Pseudomonas sp. as being located on a DNA region with two EcoRI fragments, E230 and E94 (Overhage et al. 1999).
A large number of the recalcitrant xenobiotics have been shown to be removable via the natural catabolic mechanisms of microbes, and detailed biochemical studies of the catabolic methods, together with the development of sophisticated genetic engineering, have led to the use of synthetic microbes for the bioremediation of POPs.
Taken together, our findings illustrate that molecular components of the circadian clock mechanism play a critical role in the anabolic and catabolic mechanisms of bone volume regulation.
In addition, blocking LRP-5 expression in osteoarthritic chondrocytes resulted in a significant decrease in MMP-13 expression, the basic catabolic enzyme of osteoarthritis [ 21].
Subsequent investigations have found further explanations for its anti-inflammatory and anti-catabolic mechanisms of action.
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