Sentence examples for baseline levels of inflammatory from inspiring English sources

Baseline levels of inflammatory markers for the entire study group were 110 ± 65 pg/mL for monocyte chemotactic protein-1, 0.9 ± 0.7 pg/mL for interleukin-6, and 217 ± 56 ng/mL for soluble intercellular adhesion molecule-1 (means ± standard deviation) and did not differ by treatment arm.

This study design cannot control for other day-to-day factors that could influence baseline levels of inflammatory markers.

Case subjects tended to have higher baseline levels of inflammatory markers than control subjects (Table 1), adding further justification for including baseline marker levels in each multivariate model.

A non-parametric alternative to the ANOVA statistic, the Mann–Whitney test, will be used to compare remitted and non-remitted groups in baseline levels of inflammatory cytokines and CRP if the data are not normally distributed.

As Polyzos et al. mentioned, baseline levels of inflammatory markers may not have been as relevant as those measured after baseline prior to incident diabetes, which unfortunately were not available in our study.

Comparison of baseline levels of inflammatory, endothelial and platelet markers in nondiabetic subjects and subjects with type 1 diabetes showed a significantly higher concentration of soluble P-selectin (P = 0.01) and of CD40 expression on monocytes (P = 0.006) in those with diabetes, demonstrating the chronic inflammatory response associated with diabetes.

Because the baseline levels of many inflammatory markers can differ on separate days, as was observed with sCD40L and hsCRP, this biological variability hinders the interpretation and comparison of subsequent results.

RESEARCH DESIGN AND METHODS We measured baseline levels of four inflammatory biomarkers (high-sensitivity C-reactive protein, soluble intercellular adhesion molecule-1 [sICAM-1], soluble vascular cell adhesion molecule-1, and soluble tumor necrosis factor-α receptor-1) in stored blood samples from the 1,441 participants of the Diabetes Control and Complication Trial (DCCT).

The epithelial damage that can occur in CF and the pro-inflammatory cytokines associated with infection are not present in this model, though some studies suggest that increased baseline levels of pro-inflammatory cytokines are present in CF HBEs [31, 32].

The effect of age on the baseline levels of the systemic inflammatory markers also was investigated.

Linear mixed models were used to compare the baseline levels of the systemic inflammatory markers in controls and welders and in smokers and nonsmokers.