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Scatter and bar graphs of the basal levels of plasma ACTH and cortisol in low risk (open bars) and high risk (striped bars) subjects.
As compared with controls, hypothalamic and amygdaloid CRF immunoreactivity, basal levels of plasma corticosterone and stress-induced ACTH were significantly higher in IA-treated rats.
In MCF10HER2 cells cultured without insulin basal levels of plasma membrane-localized GLUT4 were 44% higher than basal GLUT4 levels detected in the plasma membrane preparations from MCF10A cells cultured without insulin.
Notably, basal levels of plasma CORT were significantly lower in animals that had previously been exposed to EtOH during pubertal development compared with EtOH naïve animals (t(17) = 3.23, p = 0.005, Fig. 1, open bars).
Basal levels of plasma corticosterone were measured using radioimmunoassay.
Parental loss before the age of 17 years was only associated with higher basal levels of plasma cortisol in subjects who had abnormal psychiatric status as adults [ 12].
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Results showed that there was basal level of plasma s-Met in disease-free donors varying among individuals which ranged from 178.5 to 963.0 ng/mL.
The others had evidence of insulin resistance with normal plasma glucose (4.66 ± 0.30 mM) and moderate increase in basal level of plasma insulin concentration (16.8 ± 1.7 mU/L, normal <10 mU/L).
We found an increased basal level of plasma sPLA2-IIa in these controls relative to those in healthy donors, which may be a result of chronic inflammation due to fungal infection and pneumonia.
The above results demonstrated that there was basal level of plasma s-Met in disease-free donors, suggesting that, besides tumor, normal tissues also could secret s-Met into blood.
Furthermore, our data demonstrated that there was an increased basal level of plasma sPLA2-IIa in patients with SPNs relative to those in healthy donors, which may result from SPNs and chronic inflammation.
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basal levels of adrenocorticotropin
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basal levels of expression
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