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By contrast, creatine transporter deficiency cannot be treated by such supplementation, since creatine crossing of biological membranes (plasma membrane and blood-brain barrier) is dependent on its transporter.
To achieve this, the barrier is dependent not only on the crystal symmetry and interatomic bonding but also on the applied stress tensor.
As equation 11 predicts, the free energy barrier is dependent on temperature, degree of undersaturation, and by factors that affect the step edge free energy.
Severity of impact of the mycotoxin deoxynivalenol on the intestinal epithelial barrier is dependent on route of application.
The integrity and function of the epithelial barrier is dependent on the apical junctional complex (AJC) composed of tight and adherens junctions and regulated by the underlying actin filaments.
However, this is not true anymore with Complexin for which the shift of the activation energy barrier is dependent on the level of hypertonic stimulation.
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The height and shape of the fission barrier are dependent on the particular nucleus being considered.
Some barriers are dependent, some are independent and some have interrelationship.
According to the Schottky-Mott model, the Schottky barrier height is dependent on the metal work function and electron affinity of semiconductor χ (GaN χ = 4.1 eV) [1, 5, 6].
Barrier activity is dependent upon an intact transfer RNA alanine gene (cen1 tDNAAla) that is transcribed from its endogenous, centromeric location [16].
Epidermal barrier function is dependent upon changes to keratinocyte biochemistry and morphology that occur during differentiation [ 1, 2] and discrete tissue layers (Fig. 1b) are defined by histological features that reflect underlying molecular changes [ 1, 3– 7].
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