Sentence examples for balloon injury of the from inspiring English sources
The first-stage rabbit model (model 1) involved balloon injury of the aorta to validate the dose effect of external beam irradiation.
We hypothesize that HiPS-IL8RA/B-ECs transfused intravenously into rats with balloon injury of the carotid artery will target to the injured site and compete with neutrophils, thus inhibiting inflammation and neointima formation.
Young adult male Sprague-Dawley rats underwent balloon injury of the right carotid artery and received intravenous transfusion of saline vehicle, 1.5 × 10 6) HiPS-ECs, 1.5 × 10 6) HiPS-Null-ECs, or 1.5 × 10 6) HiPS-IL8RA/B-ECs immediately after endoluminal injury.
The rapid activation of ERK1/2 after balloon injury of the rat carotid artery may be associated with vascular SMC migration and proliferation in vivo [49] [51].
Male Sprague-Dawley rats (∼350 g) underwent balloon injury of the left common carotid artery as described previously.
Ad-miR-143 or Ad-miR-145 was administered to WT rats immediately after subjecting them to balloon injury of the carotid artery.
In this study, we examined the effect of the vasodilator peptide adrenomedullin on vascular remodeling induced by balloon injury of rat carotid arteries.
The role of RHAMM in smooth muscle proliferation following balloon injury of rat carotid arteries has also been reported [ 197].
We observed that following balloon injury of rat carotid arteries, VEGF levels markedly increased after 3 days, suggesting that elevation of VEGF in response to injury likely contributes to IH by suppressing later-stage apoptosis.
For the clinical application, research on the VSMC proliferation has been report in connection with restenosis, such as the development of bio-stent, the suppression of neointima formation after balloon injury, and the importance of VSMC proliferation after vascular injury and stenting [ 33- 35].
The lack of effect of the thrombin inhibitor on the cellular events during the early phase of the response to balloon injury may explain the failure of such strategies to reduce restenosis in recent clinical trials despite effects towards acute thrombotic complications.
