Sentence examples for balance of activation and from inspiring English sources

Exact(6)

Another process of sequential addition, controlled by a balance of activation and inhibition, is at work in the development of the successive molars.

At position 3, the neutral point, there is a balance of activation and repression and gene expression is at the basal level.

Thus, the balance of activation and apoptosis of autoreactive T cells (i.e., arthritis severity) seems to be controlled by TCR signal strength).

The balance of activation and detoxification is likely an important determinant of BPA effects, and this includes both maternal and fetal capabilities.

Finally, results obtained with the recently developed PG-specific TCR transgenic mice system showed that altered T cell apoptosis, the balance of activation, and apoptosis of autoreactive T cells are critical factors in the development of autoimmunity.

In addition, enzymes involved in phase I (bioactivation) and phase II (detoxification) metabolism maintain a critical balance of activation and inactivation of a wide range of chemical exposures of relevance to childhood ALL, including drugs, chemical carcinogens, insecticides, petroleum products, nitrosamines, polycyclic aromatic hydrocarbons, and other environmental pollutants [ 13].

Similar(54)

The dosimetry of benzene and its metabolites in the target tissue, bone marrow, depends on the balance of activation processes, such as enzymatic oxidation, and deactivation processes, like conjugation and excretion.

The dosimetry of benzene and its metabolites in the target tissue, bone marrow, depends on the balance of activation processes such as enzymatic oxidation and deactivation processes such as conjugation and excretion.

Significantly greater proportions of the Vδ1+ and Vδ2+ γδ T cell subsets were activated in patients with active BD; however, the balance of activation favored Vδ1+ T cells [ 31].

Because many chemicals require metabolic activation in order to exert their toxicity, the cellular distribution of the drug-metabolizing enzymes in a heterogeneous tissue, such as the lung, and the balance of metabolic activation and deactivation pathways in any particular cell are key factors in determining the cellular specificity of many pulmonary toxins.

Presumably, the genetic or mechanical changes (or both) that occur in these tissues during development must alter the balance of activation of the key Rho GTPases and favour the assembly of one actin-based machinery over another.

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