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Bacterial survival proves particularly important for bacterial propagation.
Data represents % bacterial survival (CFU) relative to initial bioburden.
Newly discovered genetic code controls bacterial survival during infections.
Fatty acid biosynthesis is essential for bacterial survival.
Inactivation of this metabolic route precludes bacterial survival.
Under these environmental conditions, acid tolerance response plays an important role in bacterial survival.
Thus, S-PRG eluate likely affects the pdhD gene, resulting in reduced bacterial survival.
A key contributing factor of bacterial survival is the ability to colonize a host effectively.
This seems to clearly confer a physiological advantage by promoting bacterial survival during sustained periods of N starvation.
The Helicobacter pylori UreI protein is not involved in urease activity but is essential for bacterial survival in vivo.
If blocking a gene kills the bug, then that gene is deemed essential for bacterial survival and could be a target for a drug.
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