Sentence examples for bacterial establishment from inspiring English sources

During the first weeks of life, the high f-calprotectin values observed in preterm infants could be linked to the gut bacterial establishment.

The latter factors are known to influence gut bacterial colonization, suggesting a possible relationship between bacterial establishment and calprotectin levels in neonatal period.

We found correlations between f-calprotectin and both microbiota per se, and other factors known to influence gut bacterial establishment as well.

In conclusion the current study demonstrates for the first time that calprotectin excretion can be linked to the gut bacterial establishment.

To determine the time course of fecal calprotectin (f-calprotectin) excretion in preterm infants from birth until hospital discharge and to identify factors influencing f-calprotectin levels in the first weeks of life, including bacterial establishment in the gut.

The aims of the current study therefore were to describe the time course of f-calprotectin excretion in preterm infants from birth through hospital discharge, and to identify factors influencing its fecal excretion during the first weeks of life, including bacterial establishment in the gut.

Investigation of biofilm structures and bacterial interaction required establishment of reliable biofilm setup protocols.

An essential feature of the pathogenicity of Salmonella is its interaction with host intestinal epithelial cells, where Salmonella invasion/entry into host cells is critical for bacterial survival and establishment of disease in a host.

Since its establishment, the bacterial community is subjected to the influence of many different factors that shape its composition within each individual.

Understanding the attachment of electro-active bacteria to electrode surfaces and their subsequent biofilm formation is one of the major challenges for the establishment of bacterial bioelectrochemial systems (BES).

RalF and DrrA/SidM function as a guanine nucleotide exchange factor of ARF, leading to the interception of host vesicle transport and establishment of bacterial replication-permissive phagosomes [13], [55], [73].