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A panel of various interacting omics approaches unraveled the specific traits of lactic acid bacteria to adapt to plants, which allow the optimal design of fermentation strategies for targeted raw matrices.
The ability of many bacteria to adapt to stressful conditions may later protect them against the same type of stress (specific adaptive response) or different types of stresses (multiple adaptive response, also termed cross-protection).
The presence of novel genetic elements containing a new methicillin- resistance gene in clinical M. caseolyticus strains from animal origin emphasizes once again the potential of bacteria to adapt to novel environments and to resist antimicrobial selective pressure of β-lactam antibiotics, which are widely used in veterinary medicine.
Consistent with a role for YeaG in allowing bacteria to adapt to adverse growth environments, a comparative analysis of virulence carried out with a S. Typhimurium ∆yeaGH and wild-type strains indicated that mutant bacteria displayed lower proliferation in the spleen (Fig. 6C), thus implying that yeaG could be a determinant the overall fitness of S. Typhimurium.
Gene transfer by natural transformation allows bacteria to adapt rapidly to changing environmental conditions.
The ability of the bacteria to adapt to changing environments is key for invasiveness.
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Because of this, analysis of the survival strategies used by this bacterium to adapt to new environmental conditions is critical for our understanding of C. burnetii pathogenicity.
Although structurally similar, the different porins have differing pore sizes, ionic selectivity and expression profiles allowing the bacterium to adapt to the variable environments [54] [56].
GIs appear to provide a critical flexible mechanism that allows a bacterium to adapt and develop increased, invasive infection in the host.
The surface localization of LPS O-antigen chains displays an interaction domain to the surrounding milieu, and its variability may be fundamental for the bacterium to adapt to changing environments within the gastric mucosa.
These four acquired PAIs encode proteins that may contribute to the overall pathogenic capacity and fitness of this bacterium to adapt to different hosts.
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