Collectively, our results support the notion that HMGB1 is an important factor, which links the APAP hepatotoxicity and gut bacterial translocation; once the enteric bacteria enter the systemic circulation, it likely triggers the systemic inflammatory response (SIRS) and leads to sepsis, MOF [ 38] and death in APAP hepatotoxicity.
Photorhabdus bacteria enter into a mutualistic symbiosis with Heterorhabditis nematodes to infect insect larvae.
Moreover, antibacterial agents and antibiotic-resistant bacteria enter the environment in different ways.
As the acids eat away at the rumen wall, bacteria enter the bloodstream and collect in the liver.
Lesions form in the root as fungi and bacteria enter damaged tissues, and root rot often occurs.
This is partly attributable to the speed of invasion as bacteria enter natural openings or wounds directly.
Bacteria enter our bodies and don't attack until they know there's enough of them to cause damage.
As a precaution against serious infection, people with artificial joints are advised to take antibiotics before dental and medical procedures that can let bacteria enter the bloodstream.
Untreated, about a quarter of those affected die because the anthrax bacteria enter the bloodstream, causing an overwhelming infection and releasing toxins.
It may be that oral bacteria enter the bloodstream, attach to fatty plaques in the coronary arteries and cause clots to form.
When the bacteria enter the bloodstream, they release a fatty substance known as an endotoxin, which injures the lining of the blood vessels and can cause fatal shock.
