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In fact it was shown that bacteria do not only communicate by direct cell-cell-contact but also via low molecular metabolites that may pass through dialysis membranes (Egland et al. (2004), Kolenbrander et al. (2010)).
We propose that bacteria do not only hitch-hike chemical pathways for altering the behavior of their host cells but they also use rapid mechanical pathways.
Nevertheless, bacteria do not only rely on their own repertoire of pTyr signalling components.
Interestingly, plant-associated bacteria do not only produce genuine plant hormones but also compounds mimicking the effect of the natural plant hormones as structural analogues.
Leaching bacteria do not only live in perfect symbiotic association in leaching environment; they may have a competition between them leading to changes in microbial population composition overtime.
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This is not so bad because usually these bacteria do not cause disease, and they only have one or two resistance genes each.
Bacteria do not change the direction of motion only due to tumbling, but also due to collisions with the surrounding medium, which are governed by thermal fluctuations.
Though the average bacterium has fewer than 5,000 genes, compared with around 20,000 protein-coding genes in the average mammal (bacteria do not go in much for RNA-only genes), there are lots of species of bacteria.
The observed differences in the number of transcriptional regulators involved in carbohydrate metabolism may also reflect the fact that pathogenic bacteria do not require a versatile sugar metabolism since only a limited range of carbohydrate nutrients might be present in their natural habitats.
While carbohydrates are a common energy source for eukaryotes, these molecules are metabolized by only a limited number of species of bacteria, since most bacteria do not possess the necessary enzymes to metabolize these often complex molecules.
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