The phrase "axis participates" is grammatically correct and can be used in written English.
It can be used in contexts where you are discussing the involvement or role of an axis in a particular process or system.
Example: "In the analysis of the data, it is crucial to understand how the axis participates in the overall dynamics of the model."
Alternatives: "axis is involved" or "axis plays a role".
Such observation strengthens the hypothesis that CXCR4/CXCL12 axis participates in the promotion of in vivo NB cell proliferation, together with other micro environmental factors, which need to be identified.
Together, these data suggest that the Noxa/Mcl-1 axis participates in apoptosis initiated by overall nutrient stress.
As a result, the SDF-1/CXCR4 axis participates in directing the metastasis of these cells to bones that express high levels of SDF-1 [ 22].
Several previous studies also provide evidence that the β-catenin/TCF axis participates in pancreatic cell proliferation and differentiation [ 127– 127].
The SDF-1 α-CXCR4 axis participates in the recruitment of endothelial progenitor cells (EPCs) from bone marrow to areas of vascular damage, constituting an important mechanism of vascular repair [ 52, 53].
Although the HPA axis participates in the sickness response (Lenczowski et al., 1997), the current results indicate that the HPA axis did not contribute to the aggravation of sickness by combined NLR + TLR agonism, since neither FK565 nor MDP augmented the LPS-induced rise of plasma corticosterone, both in the absence of stress and following exposure to tail suspension stress.
Overall, it is important to note that many of the major mediators of the stress response and melancholic depression, such as the subgenual prefrontal cortex, the amygdala, the noradrenergic system and the CRH/HPA axis participate in multiple reinforcing positive feedback loops.
We attempted to further determine whether the JNK ATF2 axis participated in the regulation of cell cycle progression and apoptosis.
Our study partly confirmed the previous conclusions that the CCL2 CCR2 axis participate in the modulation of Nav1.8 expression and current density.
Our study defines the NFAT4/ miR-324-5p/Mtfr1 axis, which participates in the regulation of mitochondrial fission and cardiomyocyte apoptosis, and suggests potential new treatment avenues for cardiac diseases.
The CXCL12/CXCR4 axis also participates in the recruitment of inflammatory cells as shown in animal models of allergic airway disease and rheumatoid arthritis (RA) [ 13- 15].
