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In M. oryzae, nitrogen availability appears to regulate pathogenicity.
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Kir4.1 and the CaSR have recently been shown to physically interact in human embryonic kidney cells and in kidney homogenates, and the CaSR appears to regulate Kir4.1 activity by decreasing Kir4.1 membrane availability via a Gα- and caveolin-dependent pathway [ 75].
A feedback mechanism involving oxygen delivery to the tissues appears to regulate EPO production [9].
The role of mitochondrial EGFR remains poorly understood but it appears to regulate apoptosis and autophagy.
Thus DG appears to regulate R cell elongation.
SF-1 appears to regulate cell growth via multiple mechanisms.
Dimerization appears to regulate surface trafficking of TMEM16A.
As CTK regulates meristem activity in leaves [ 38], miR396k appears to regulate soybean leaf meristem activity.
Variants of the guanosine triphosphate cyclohydrolase 1/dopa-responsive dystonia gene appear to regulate nociception.
miRNAs appear to regulate cardiomyocyte apoptosis through multiple mechanisms.
NdtA does not appear to regulate xprG expression.
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