This briefly outlined process is structural autoregulation as described in detail by Folkow [ 2].
In addition, other posttranscriptional mechanisms of SRSF1 autoregulation were described such as nuclear retention of alternative SRSF1 transcript variants or regulation of the translational efficiency of its transcripts [ 11, 12].
Both positive and negative autoregulation has been described for T-box genes in ascidians and humans, suggesting autoregulation may be a common feature of this gene family (Conlon et al. 1996; Takahashi et al. 1999; Mitani et al. 2001; Sun et al. 2004; Imai et al. 2006; Andreou et al. 2007).
Furthermore, impaired autoregulation has been described in some brain-injured areas, in some TH-treated patients, and inconstantly in CA patients.
This suggests that similar mechanisms of autoregulation to those described for let-7 and lin-4 in C. elegans might exist for mir-5364 in the parasite.
An impaired metabolic cerebrovascular autoregulation [1] was described as a causative factor in PSH, and vasospasm was seen on angiography.
This "phase-led property" of cerebral autoregulation has been previously described with CBFV changes preceding BP changes [ 14].
The presence of W-boxes in the TaWRKY53 and OsWRKY53 promoters implies either regulation by other WRKY transcription factors (Rushton et al. 2010; Eulgem and Somssich 2007), or autoregulation as has been described for the orthologs AtWRKY33 in Arabidopsis (Mao et al. 2011) and PcWRKY1 in parsley (Petroselinum crispum L). (Turck et al. 2004).
In addition, we allow STAT3 autoregulation that implicitly describes several possible feedback mechanisms (e.g. amplification through IL21 signals and positive feedback loops through other genes [ 4, 6]).
Cerebrovascular autoregulation was assessed by calculating a moving correlation coefficient (the index of cerebrovascular autoregulation, Mx) between MAP and FV as described previously [ 19].
