The phrase "autoimmune element" is correct and usable in written English.
It can be used in contexts discussing aspects related to autoimmune diseases or conditions, particularly when referring to specific factors or components involved.
Example: "Researchers are investigating the autoimmune element that triggers the body's immune response against its own tissues."
Alternatives: "autoimmune factor" or "autoimmune component".
A significant body of evidence now links aberrant epigenetic regulation of gene expression with rheumatic disease and points toward the use of epigenetic targeting agents as potential new treatment options, particularly for those conditions associated with an autoimmune element.
Although an underlying "occult" autoimmune element is suggested in the provoked A−β+ subtype by progressive β-cell loss and the presence of type 1 diabetes associated HLA susceptibility alleles, the unprovoked A−β+ subtype could represent a truly nonautoimmune syndrome of KPD.
Dysregulation of the HLA/T-cell receptor axis seen here reminds us of autoimmune elements seen in other inflammatory illnesses, such as CD.
Disease-associated T-cell autoreactivities are seen in most type 1 diabetic patients and are thought to emerge before islet autoantibodies, but host factors that impact autoimmune elements remain uncertain.
Notably, autoimmune regulatory element-binding factors (V$AIRE) and PPAR (or V$PERO) were conserved in these promoters.
Wide coverage of self structures is ensured by the random expression of peripheral tissue antigens under the control of the autoimmune regulator (AIRE) element and other less well characterized mechanisms [ 21, 22].
IL-17 is one such pro-inflammatory cytokine which is a key element associated with autoimmune diseases, including SLE.
BBB breakdown is an important pathology commonly associated with AD, as it allows for the extravasation of potentially damaging humoral elements such as autoimmune antibodies, complement components, A β42, and proinflammatory mediators [ 7, 50– 50].
Although this model has contributed to understanding the immune component of autoimmune attacks on myelin, it is somewhat harder to dissect out elements of demyelination and remyelination that are essential central nervous system (CNS) responses.
The rationale made sense: I had this autoimmune condition, in which one immune element, T.N.F.-alpha, was running amok in me for reasons unknown.
Autoreactive T cells are a central element in many systemic autoimmune diseases.
