Sentence examples for augmented susceptibility from inspiring English sources

Increased atherogenic potential has been ascribed to these particle because they seem to have reduced affinity to LDL receptors, increased propensity for transport into the vascular wall, increased binding to arterial wall proteoglycans, and augmented susceptibility to oxidative modifications (25).

These results suggest that in diabetic mice undergoing polymicrobial infection, mast cells play a key role in the neutrophil migration failure due to reduction of the CXCR2 expression, resulting in bacterial spreading and systemic release of mediators, and as a consequence augmented susceptibility to sepsis development.

3. Environmentally or genetically determined increases in Cu levels might augment susceptibility to infection with intracellular pathogens, by causing a decrease in IFN-γ production.

Plasma Cu positively correlates to (non-protective) humoral immune response across patient groups, and reciprocally, increased Cu levels decreased in vitro (protective) IFN-γ production, implying that environmentally or genetically determined increases in Cu levels might augment susceptibility to infection with intracellular pathogens.

For example, Chakraborty et al. found that irradiated cancer cells highly express Fas receptor and ICAM-1 in a dose-dependent manner, and phenotypic modification augmented the susceptibility of cancer cells to CTLs [ 19].

These data suggested that type III EBV latency in B lymphoma cells augments the susceptibility to MV-induced cytolysis, although the magnitude of augmentation varies among cell types and the MV strains.

We could show that this defect markedly augments the susceptibility to pulmonary edema of these mice.

Their cytotoxicity increases during exercise, but it is depressed after exercise, probably augmenting the susceptibility to infections [ 10– 10].

To augment routine susceptibility studies performed at MDCH, a special surveillance project to collect gonococcal isolates from clinical laboratories across the state was initiated in July 2002 and continued through September 2004.

In our study, we observed an increased chemical-related sensitivity in subjects with homozygous GSTT1 deletion leading to the assumption that the deletion of this gene augments the susceptibility to environmental chemicals.

As analyzed by EBV-positive and -negative counterparts of the same cytogenetic background, type III EBV latency, not type I, was shown to augment the susceptibility of B lymphoma cells to MV-induced cytolysis.