Sentence examples for augmented resistance from inspiring English sources

PTPRS deletion was associated with elevated EGFR phosphorylation in tumor tissues, and PTPRS knockdown augmented resistance to erlotinib in head and neck squamous cell carcinomas cell lines.

In A. aceti, overexpression of the groESL operon led to augmented resistance to acetic acid [ 30], which may be explained by the fact that chaperonins protect proteins under denaturing conditions such as low pH [ 31].

Splenomegaly is related not only to blood congestion as a consequence of increased portal pressure and augmented resistance to splenic vein outflow but also to multiple other histopathologic changes, such as arterial aneurysms [ 22] and concern with endothelin [ 23], and the immunologic pathway from the liver [ 24].

Overall, curcumin provided Drosophila with augmented resistance to overcome radiation-induced oxidative stresses.

Our results demonstrate that S180 only enhances resistance against LL/2 locally in SR/CR mice and argues that the ability of S180 to induce leukocyte infiltration is the critical event in augmenting resistance.

Furthermore, the deregulation of signal transduction pathways, resulting from oncogenic mutations, can lead to androgen independent proliferation of cancer cells therefore augmenting resistance to AR-targeted therapies (4, 5).

The lack of adequate moisturizing may thicken the secretions, which augments resistance to the passage of air, reduces the gas exchange effectiveness, and increases the risk of respiratory infections [ 10].

41 Adult worms with an increased daf-16 activity live longer than control worms and appear to possess augmented stress resistance, including resistance to ROS. 42 Indeed, deletion of FoxO transcription factors (FoxO1, FoxO3, FoxO4) decreases the expression of ROS-removing enzymes such as superoxide dismutase 2 and accumulates ROS.

Traditionally, identification and treatment of augmented coronary resistance have focused on obstructive atherosclerosis of the epicardial arteries.

However, over the past two decades it has become increasingly apparent that augmented vascular resistance may also reside in the coronary microcirculation in a number of clinical conditions [ 13, 39].

The increased rigidity and adhesiveness of P. falciparum-infected erythrocytes result in augmented haemodynamic resistance in the microvasculature (Raventos et al., 1985) and play an important role in the pathogenesis of malaria.