Sentence examples for augmented permeability from inspiring English sources

CSE in presence of HG produced a synergistic increase in release of vascular endothelial growth factor that was accompanied by decreased TEER and augmented permeability to labeled dextrans in a size-dependent manner.

Upon exposure to NECA or Lexiscan, monolayers of Bend3 mouse brain endothelial cells (CD73+, A1 AR+, A2A AR+) lowered their transendothelial cell electrical resistance, a phenomenon known to be associated with increased paracellular space and augmented permeability [ 62, 63].

11 In intestinal inflammation, as a result of mucosal ulceration or augmented permeability, the extravasion of AAT into the gut can be measured in the stool and is the classic fecal marker for protein losing enteropathies.

Another theory that might explain the findings of the study is that of the augmented permeability of the blood brain barrier in patients with neurodegenerative diseases, such as multiple sclerosis [ 32, 33]; this phenomenon may give to the H.pylori the chance to enter the barrier and accelerate or influence their course.

Hence, at least two factors interact to determine the development of pulmonary oedema, namely loss of the tissue safety factor and augmented microvascular permeability [ 27].

Experimental recruitment of ARs either by the broad spectrum agonist NECA or the engagement of both A1 and A2A receptors by selective agonists (CCPA and CGS21680) cumulatively and transiently augmented BBB permeability facilitating the entry of intravenously infused macromolecules (including immunoglobulins such as the anti-β-amyloid 6E10 antibody) into the CNS [ 27].

Probably MMP2 and MMP9 augment pleural permeability via other mechanisms.

Then, we discuss methods to augment conventional "permeability and retention" effects for macromolecular or nanosized bioconjugates in cancer tissue.

Several treatments aimed to increase cAMP (forskolin) rapidly induced redistribution of AQP8 to the plasma membrane, augmenting water permeability [ 38].

These data suggest that Pb exposure selectively alters the cellular level of claudin 1, which, in turn, reduces the tightness and augments the permeability of tight BCB.

In line with this hypothesis, induction of AR signaling by selective agonists efficiently augments BBB permeability in a transient manner and promotes the entry of macromolecules into the CNS.