The phrase "atypical complex" is correct and usable in written English.
It can be used to describe something that is unusual or not typical in its complexity, often in academic or analytical contexts.
Example: "The study revealed an atypical complex of symptoms that did not fit the standard diagnostic criteria."
Alternatives: "unusual complexity" or "nonstandard complexity".
The objective of the study was to evaluate the regression, relapse, and live birth rates of early-stage endometrial cancer (EC) and atypical complex hyperplasia (ACH) with fertility-sparing treatment.
Associated atypical complex hyperplasia was found in 22 cases.
Endometrioid endometrial carcinoma (EEC), or Type 1 cancers, are oestrogen dependent, often develop in a background of atypical complex hyperplasia and account for over 75% of cases.
These features were in keeping with either atypical complex hyperplasia within an endometrial polyp associated with metaplastic changes, or a polypoid uterine teratoma.
Furthermore, there is a progressive increase in microvessel density from benign endometrium through the cancer precursor, atypical complex hyperplasia (ACH) to invasive disease (Abulafia et al, 1995).
Patients with normal endometrium (n = 20), endometrial hyperplasia (n = 10) (simple, n = 1; complex, n = 2; atypical complex, n = 7), adenocarcinoma and carcinosarcoma (n = 134) were treated at Okayama University Hospital between January 2000 and November 2009.
At 30 weeks, incidences of atypical and complex endometrial hyperplasia were significantly decreased by danazol-treatment.
Single or small clusters of cells and atypical or complex glandular arrangements that are beyond normal mucosal architecture were detected more frequently in the 7 high-consensus tumors.
Moreover, recognition of ADEs by the treating physicians is challenging in elderly patients because their disease presentation is often atypical and complex.
An atypical PRC1 complex, dRAF, comprising the proteins dRING, PSC, and the histone demethylase KDM2 has been identified in Drosophila (Lagarou et al., 2008).
Our data also suggest that MIG-2/RhoG, which activates the Dock/ELMO atypical RacGEF complex in human and C. elegans cell corpse engulfment (deBakker et al. 2004), does not activate the CED-2/ 5/ 12 Dock/ELMO RacGEF complex in DTC migration.
