The phrase "attenuate the induction" is correct and usable in written English.
It can be used in contexts related to reducing or lessening the effects of an induction process, often in scientific or technical discussions.
Example: "To improve the efficiency of the system, we need to find a way to attenuate the induction of electromagnetic interference."
Alternatives: "reduce the induction" or "diminish the induction".
This biocompatible implant can deliver HCQ above therapeutic concentrations in a controlled manner, reduce submucosal immune cell recruitment, improve mucosal epithelium integrity, decrease protein and gene expression of T-cell activation markers, and attenuate the induction of key pro-inflammatory mediators.
Analogous to the findings following interruption of CREB expression (Fig. 7B), CBP siRNA could also significantly attenuate the induction of IL-6 (Fig. 8D).
The conventional role of macrophage in tumor rejection through recognition of tumor antigen and participation in induction of anti-tumor T cell response is changed at the tumor site where it seems to produce elevated levels of immunosuppressive cytokines like IL-10 and TGF-β that effectively attenuate the induction of anti tumor response.
It has been shown that NF-κB inhibitors attenuate the induction of renal inflammation and injury in animal models [ 95, 96].
It is known that mechanisms that inhibit or attenuate the induction of cancer cell apoptosis produce resistance to certain antitumor drugs [ 20].
Activated TGF- β in the CNS may inhibit the proliferation and activation of these spinal glial cells to attenuate the induction of neuropathic pain [ 188– 188].
The PEGylation of conventional liposomes attenuated the induction of the phenomenon somewhat with increasing molar content of PEG derivative and PEG chain length.
In contrast, RNAi-mediated knockdown of BAF60a in the liver attenuates the induction of genes involved in FAO in response to starvation, leading to TG accumulation and liver steatosis.
Thus, knocking down the expression of SREBP1c greatly attenuated the induction of CD36 caused by ritonavir.
Similarly, treatment with either E2 or the ERβ agonist attenuated the induction of the intestinal iNOS response in OVX rats subjected to T/HS whereas the ERα agonist was only partially protective.
Conversely, silencing of NR2F2 expression with a NR2F2 siRNA attenuated the induction of TFAP2A expression during CTB cell differentiation as well as the expression of other genes that are specific markers of STB differentiation.
