The phrase "attachment of pathogens" is correct and usable in written English.
It can be used in contexts related to biology, microbiology, or medicine, particularly when discussing how pathogens adhere to surfaces or host organisms.
Example: "The attachment of pathogens to host cells is a critical step in the infection process."
Alternatives: "adherence of pathogens" or "binding of pathogens".
Cranberry, long advocated for prevention of acute cystitis, contains proanthocyanidines inhibiting the attachment of pathogens to uroepithelium [27].
The critical first step of a microbial infection is usually the attachment of pathogens to host cell glycans.
Lactobacilli have been proven to be effective in inhibiting enteropathogenic infections [ 3, 4] partly by competition with enteric pathogens for enterocyte binding sites, subsequently blocking the attachment of pathogens to IECs [ 5]. Cell surface properties, such as hydrophobicity and aggregation (autoaggregation and coaggregation), are necessary for the adhesion of probiotics.
Some human milk oligosaccharides have been shown to directly bind to pathogenic bacteria, inhibiting the attachment of these pathogens to host cells [ 6].
The attachment of these pathogens to foodstuff and food-contact surfaces is an important mechanism in the human contamination process.
For example, aggregative fimbriae have a role in binding and invading epithelial cells in Escherichia coli and S. enterica [28], and are also implicated in the attachment of these pathogens to alfalfa sprouts [11], [29], [30].
These properties enable the two strains to inhibit the attachment of some pathogens to IECs.
L. salivarius inhibited the attachment of three pathogens (25.0% to 38.2%) except for S. typhimurium (1.7%).
The attachment of enteric pathogens to host intestinal epithelial cells (IECs) is an important cause of pathogenesis.
Several studies have demonstrated the inhibition of the attachment of enteric pathogens such as E. coli and Campylobacter jejuni and noroviruses with HMO [ 151].
The intimate attachment of A/E pathogens to enterocyte surface is mediated through interactions of the bacterial outer membrane protein intimin and the translocated intimin receptor (Tir) [ 7].
