The phrase "atrial system" is correct and usable in written English.
It can be used in contexts related to anatomy, physiology, or medical discussions, particularly when referring to the heart's atria or a system involving atrial structures.
Example: "The atrial system plays a crucial role in the heart's ability to pump blood efficiently."
Alternatives: "atrium system" or "heart atrial network".
We hypothesize here that the source-sink SAN-atrial system is set, in physiological conditions, at a given value of (S, R J ), say (11, 590 MΩ); that is, 11 SAN cells are spatially arranged around a single atrial cell and are cumulatively coupled to it with a total of 34 gap junctional channels (assumed to have, as pointed out above, a single channel conductance of 50 pS).
The condition, while not harmful in and of itself, is usually an indication of problems with the atrial conduction system and frequently results in the development of atrial fibrillation.
Studies had shown that smoking increases conduction velocities, and decreases effective refractory period by decreasing parasympathetic tone in atrial conduction system and also increases sympathetic tone which could be responsible for the excess SVEs.
Conversely, implanted devices collect important diagnostic data (eg, atrial fibrillation onset, system integrity) that remain concealed between device interrogations.
Along with the degradation of the atrial T-tubular system, HF also caused altered Ca2+ cycling.
Factors that conferred increased risk of appropriate shock included a history of nonsustained VT, atrial fibrillation, pacemaker system in situ, and higher creatinine concentration.
Canine and human studies [ 40– 44] have revealed an extensive distributed system of atrial pacemakers, the atrial pacemaker complex, which extends well beyond the anatomically defined SAN and includes primary and subsidiary pacemakers located within the right atrium.
Recent evidence has linked activity of the autonomic nervous system and atrial electrical properties with the triggering of atrial arrhythmias such as atrial fibrillation (AF) and flutter (Arora 2012; Lo et al. 2011; Park et al. 2012).
Functionally distinct conductive tissues of the heart, such as the nodes and bundles of the conduction system and atrial and ventricular myocardium, express different combinations of the three cardiac connexins and are interconnected by gap junctions in specific three-dimensional patterns that are consistent with the anisotropic conduction properties of each tissue.
LMNA-related DCM is characterized by early onset of atrial fibrillation and conduction system disease, and subsequent progression to sudden cardiac death and heart failure [ 7, 8].
It has been proposed that, because of the lack of a regular T-tubule system in atrial myocytes, differential spatial distribution of LTCCs with regard to their coupling to RyR2s may underlie a unique atrial myocyte Ca2+-signaling process.
