The phrase "at receptor" is correct and usable in written English.
It can be used in scientific or medical contexts, particularly when discussing interactions at a cellular or molecular level.
Example: "The drug binds specifically at receptor sites, influencing cellular activity."
Alternatives: "on receptor" or "in receptor".
Angiotensin II interacts with specific cell surface angiotensin AT1 and AT2 receptors and, in some vertebrates, with an atypical angiotensin AT receptor.
The purpose of this study was to evaluate the feasibility of noninvasive imaging of angiotensin II (AT) receptor upregulation in a mouse model of post-myocardial infarction (MI) heart failure (HF).
The properties of urotensin II (U-II) receptor (UT receptor) and angiotensin II (ANG II) receptor (AT receptor) in primary human skeletal myoblasts (HSMM) and differentiated skeletal myotubes (HSMMT) were characterized.
Understanding of related general issues, e.g. concurrent medication and preparation of the patient for the study, including the use of pharmaceutical interventions (e.g. ACEI, AT receptor blockade, furosemide).
Moreover, BNGR-A16 responded to AT specifically and dose-dependently (Figure 4B), showing that this receptor is the functional AT receptor.
Finally, double staining using immunohistochemistry and in situ hybridization revealed AT receptor expression in the sNPF-producing cells in the CC (Figure 6C).
Fujino, M. et al. A small difference in the molecular structure of angiotensin II receptor blockers induces AT receptor-dependent and -independent beneficial effects.
These experiments indicate that the sequence of the tri-nucleotide spacer is non-random at receptor-bound genomic loci, influences ERα-DNA-binding affinity, and modulates transactivation potential of the receptor ligand DNA complex.
AA residues at receptor-binding site of HA protein were conserved among the 35 H9N2 AIVs.
Tetrodotoxin blocks Na+ conductance by binding extracellularly at receptor-site 1 of Nav channels preventing monovalent cations from accessing the pore.
The saturation of the effect at receptor-relevant concentrations (Fig. 3) and the ability of the potent prostamide antagonist, AGN 211336, with a pA2 of 7.6 against the prostamide receptor (20), to block the stimulatory effects of bimatoprost on both hair growth parameters in this dynamic bioassay (Fig. 3) also indicate a receptor-mediated response.
