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An imbalance or dysfunction of adipose tissue (AT) contributes to obesity-induced chronic inflammation, which in turn results in energy metabolism disorders such as insulin resistance (IR), type 2 diabetes, inflammation and cardiovascular disease [1] [11].
Adipose tissue (AT) contributes to metabolic dysfunction through imbalanced production of adipokines, including cytokines.
The former model corresponds to cases when parental genomes make equal contribution to the SN transcriptiome, whereas the latter model assumes that the tetraploid parent (TC) contributes two parts and diploid parent (AT) contributes only one part to total gene expression.
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AT contributed Section 4.2.
AT contributed to the radiological analysis of the CBCT measurements and the preparation of the manuscript.
AT: contributed to statistical analysis.
AT contributed to the study design and data collection.
AT contributed to the study design, performed some of the statistical analysis, and drafted the manuscript.
AT contributed to study design, data interpretation, article revision and review of the final manuscript.
AT: contributed to this work by writing the paper and performing the literature research.
AT contributed to the study design and collection of data, and critically reviewed the first draft of the paper.
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