The phrase "associated impairment" is correct and usable in written English.
It can be used in contexts discussing conditions or issues that are linked to a particular impairment or disability.
Example: "The study focused on the cognitive effects of aging and the associated impairment in memory function."
Alternatives: "related disability" or "connected impairment".
Childrens' and parents' reports differed in regard to the association between reported symptom scores and associated impairment with children reporting higher impairment due to emotional problems.
Adaptations in the NF-κB system may be essential for persistent neuroplastic changes and neurodegeneration underlying dependence to alcohol and associated impairment of cognitive functions.
By both parent and self-report, youths with anxiety disorders exhibit significantly more irritability and associated impairment than healthy subjects.
The most commonly reported impact was on eating and the most commonly associated impairment with this was toothache followed by oral ulceration.
Despite an increase in knowledge about the epidemiology of intimate partner violence (IPV), much less is known about interventions to reduce IPV and its associated impairment.
The DSM-IV symptom criteria of behavioral diagnoses often require associated impairment at a clinically significant level (e.g., aggression toward other people).
Reliable information on headache characteristics, including frequency and intensity, headache-associated impairment, and use of analgesic medications, may depend on the assessment method used.
Delirium occurs commonly in settings of palliative care (PC), in which patient vulnerability in the unique context of end-of-life care and delirium-associated impairment of decision-making capacity may together present many ethical challenges.
Finch showed that TAT gene expression is reduced in old mice relative to young mice; however, when he injected glucocorticoids directly into old and young rodents, he found no age-associated impairment in their responses.
Therefore, we have attempted to determine the mechanisms underlying the HBV-associated impairment of insulin signaling.
Overexpression of the cytosolic superoxide dismutase (SOD1), or the extracellular SOD (EC-SOD), but not the mitochondrial MnSOD (SOD2), reverses age-associated impairment of long-term potentiation (LTP), and prevents loss of spatial memory[16].
