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Here we assessed whether the levels of certain stem cells may predict the progression of Duchenne muscular dystrophy (DMD).
First, we assessed whether the levels of hypophosphorylated heavy neurofilaments (NF-H), as detected with the monoclonal antibody SMI32, were altered.
As p75NTR signaling has also been implicated with tau hyperphosphorylation in isolated hippocampal neurons (Saez et al., 2006), we assessed whether the levels of the above kinases changed in relation to the expression of p75NTR itself.
As one of the main functions of FcγRIIb on B cells is to control the development of autoimmunity by providing feedback inhibition in order to limit the secretion of autoantibodies, we assessed whether the levels of autoantibodies on RA patients were related to the expression of this inhibitory receptor on B cells.
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We assessed whether the level of hypoxia was altered in the tumours.
We also assessed whether the level of the slowest-migrating band was also sensitive to the addition of LiCl (10 mM, 24 hrs) to the culture medium.
Additionally, we assessed whether the level of kif21b expression correlated with the extent of grey matter demyelination in MS patients.
We assessed whether the level of E10 inclusion in tau RNA could be reduced by trans-splicing in the context of FTDP-17 mutations.
This study assessed whether the level of circulating methylated GSTP1 (mGSTP1) in plasma DNA is associated with chemotherapy response and overall survival (OS).
Next, we assessed, whether the level of viral load within the HVL group is directly or inversely correlated with host cellular factor expression, and results indicate that none of the transcripts significantly correlate with either CD4 count or nadir CD4 count.
In the meantime, the OFT has been collecting information from the banks to allow it to assess whether the levels of their charges are unfair.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com