The phrase "assessed binding of" is correct and usable in written English.
It can be used in scientific or technical contexts, particularly in research related to molecular biology, chemistry, or pharmacology, where the binding affinity of molecules is evaluated.
Example: "The researchers assessed binding of the drug to its target receptor to determine its efficacy."
Alternatives: "evaluated the binding of" or "measured the binding of".
Since TESS predicted that Sp1 binds to MCRE, we assessed binding of cellular Sp1 to MCRE by electrophoretic mobility shift assay (EMSA).
Two mechanisms were assessed: binding of the tracer to melanin or to sigma receptors of melanoma cells.
Next we assessed binding of recombinant SNAP25 and VAMP2 to synatxin1B immunopurified from mouse brains.
To validate the binding of NLX to FLNA, we assessed binding of [3H]NLX to membranes prepared from the human melanoma cell line M2 that lacks filamin and to membranes from its FLNA-transfected subclone A7.
Because AcH4 and H3K4me3 function to enhance gene transcription, in part, by facilitating binding of RNA polymerase II to promoters of regulated genes, we next assessed binding of RNA polymerase II to the TNF-α promoter at both 2 and 4 hrs post LPS treatment in sham and CLP macrophages.
We assessed binding of serum IgG to 3 in the absence of a competitor, or in the presence of 500 nM insulin or GAD65.
To assess binding of KatA to monomeric or polymeric Vn, 50 nM Vn was coated to Polysorb plates as described above.
To confirm the DNA-binding data, we employed chromatin immunoprecipitation (ChIP) assays to assess binding of Stat1 and Stat3 to the Cdkn1b promoter in vivo (Fig. 3B).
To assess binding of CPS to LL-37 in the presence of human CD14 and LBP, an EMSA assay using native gel conditions was performed [18].
To assess binding of recombinant HcpA for CFH and CFHR-1, ligand affinity blotting techniques and ELISA were employed in combination with intact HcpA and various deletions thereof.
To test this hypothesis, we performed ChIP to assess binding of ESE3 to the promoters in parental and ERG-expressing LNCaP cells.
